Belly fat, especially visceral fat around internal organs, can damage the digestive tract and raise gallbladder risk. Belly obesity and gallbladder health are linked by many physiological pathways.
Bile and Gallbladder Function: The gallbladder stores bile, a liver-produced digesting fluid. Bile helps the small intestine breakdown and absorb lipids. Belly fat affects gallbladder function and bile production.
Increased Gallstone Risk: Visceral fat releases chemicals that might cause gallstones from metabolic activity. Gallstones, solid particles in the gallbladder, can cause inflammation (cholecystitis) and bile duct blockage.
Insulin Resistance and Metabolic Factors: Excess belly fat is commonly linked to insulin resistance, a condition where cells become less insulin-responsive.
Inflammatory Factors: Visceral fat can cause systemic inflammation by producing inflammatory chemicals. Chronic inflammation can impair gallbladder function and raise risk of complications.
Adipose tissue, particularly visceral fat, is hormonally active. Extra abdominal fat might cause hormonal imbalances that compromise gallbladder function. Obesity-related estrogen dominance increases gallstone risk.
Abdominal Organ Pressure: Belly obesity can strain neighboring organs, especially the gallbladder. Increased abdominal pressure can disrupt bile flow and cause gallbladder malfunction.
Excess belly fat and gallbladder damage can cause abdominal pain, bloating, and indigestion. In severe cases, gallbladder inflammation or bile duct obstruction may occur.
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